Endogenous sex hormones and the development of type 2 diabetes in older men and women: the Rancho Bernardo study.
By Jee-young Oh, E. Barrett-Connor, Nicole M Wedick, D. Wingard. Diabetes Care, 2002
624 citations recorded by the source corpus.
Subfields: Endocrinology, Diabetes and Metabolism, Psychiatry and Mental health
View Paper: https://www.semanticscholar.org/paper/dcf44523fc119ac1c66193164714a89898784ca0
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Endocrinology, Diabetes and Metabolism
Citations in the corpus
- Sex differences of endogenous sex hormones and risk of type 2 diabetes: a systematic review and meta-analysis. (2006, background): “However, no prospective studies directly link PCOS with risk of incident type 2 diabetes, and PCOS is unlikely to explain the testosterone association among postmenopausal women and populations of diabetic cases and controls both with PCOS.(6,8,10,13,14,60) Furthermore, reverse causation explanation”
- Low serum testosterone and mortality in older men. (2008, background): “We, and others, have shown that low testosterone precedes the development of central obesity (24, 25), the metabolic syndrome (26, 27), and diabetes (6, 27, 28) over the following 10–15 yr in nonobese men without these conditions at baseline.”
- Testosterone and sex hormone-binding globulin predict the metabolic syndrome and diabetes in middle-aged men. (2004, background): “In 294 middle-aged and elderly men, total testosterone levels were lower in the 26 men who developed diabetes during the 8-year follow-up, but adjustment was made for only age, BMI, and systolic blood pressure (19).”
- Effect of oestrogen plus progestin on the incidence of diabetes in postmenopausal women: results from the Women’s Health Initiative Hormone Trial (2004, background): “In a prospective study of older women who were not using hormone therapy, endogenous levels of bioavailable oestradiol and testosterone were positively associated with 8-year follow-up levels of fasting insulin and calculated HOMA-IR, while only bioavailable testosterone predicted incident diabetes ”