Homocysteine and atherothrombosis

By Erling Falk, Ji Zhou, J. Møller. Lipids, 2001

877 citations recorded by the source corpus.

Subfields: Cardiology and Cardiovascular Medicine, Rheumatology, Surgery

View Paper: https://www.semanticscholar.org/paper/dba5fc029e42b69e09c26fb1868cbedc506f6bac

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1. Mark P. Mattson
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Citations in the corpus

• Guidelines for the Primary Prevention of Stroke (2011, neutral): “a point mutation in which cytosine is replaced by thymidine at position 677 (MTHFR C677T).(759) Hyperhomocysteinemia also is caused by nutritional deficiencies of pyridoxine (vita-”
• Homocysteine Elicits a DNA Damage Response in Neurons That Promotes Apoptosis and Hypersensitivity to Excitotoxicity (2000, methodology): “The normal range of homocysteine concentrations in plasma is 5–15 mM, and levels of homocysteine in CSF and brain tissue are reported to range from 0.5 to 10 mM (Welch and Loscalzo, 1998).”
• Endoplasmic reticulum stress signaling in disease. (2006, result): “Homocysteinemia is associated with the development of atherosclerosis, certainly in patients with rare inherited disorders of amino acid metabolism, but also in the wider population, as indicated by large epidemiological studies (42, 70, 201).”