Complement evasion by human pathogens

By John D Lambris, D. Ricklin, B. Geisbrecht. Nature Reviews Microbiology, 2008

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Pathogenicity and virulence of Staphylococcus aureus (2021, background): “convertases from one of three independent pathways (the lectin classical, and alternative pathways) [99].”
Complement regulators and inhibitory proteins (2009, background): “By contrast, tumour cells and pathogenic microorganisms attach host complement regulators and mimic a self surface zone to escape complement surveillance, resulting in unrestricted growth and infections, respectivel”
Complement System Part II: Role in Immunity (2015, background): “These mechanisms of complement evasion can be classified in different groups (157) (Figure 9).”
The Complement System (2010, background): “…complement activation, including regulation of activation, amplification, opsonization, phagocytosis, chemoattraction, cell lysis (for a review, see Lambris et al. 2008; Rooijakkers and van Strijp 2007) by either proteolysis of complement proteins or mimicking the action of complement proteins or…”
Complement - a key system for immune surveillance and homeostasis (2010, background): “In fact, many pathogens are protected from lysis through their cell wall architecture (for example, Gram-positive bacteria) or by employing evasive strategies that interfere with TCC assembl”