Mycobacterium tuberculosis: here today, and here tomorrow

By D. Russell. Nature reviews. Molecular cell biology, 2001

825 citations recorded by the source corpus.

Subfields: Epidemiology, Infectious Diseases, Surgery

View Paper: https://www.semanticscholar.org/paper/d885be3d056a308c3e961b8117d2ebdb0a44b970

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Epidemiology

1. M. Gengenbacher
2. Olivier Schwartz
3. Stefan H. E. Kaufmann
4. B. B. Finlay
5. G. McFadden
6. Paul D. Cotter
7. C. Hill
8. R. Medzhitov

In Infectious Diseases

1. M. Gengenbacher
2. Stefan H. E. Kaufmann
3. B. B. Finlay
4. C. Nathan
5. G. McFadden
6. Paul D. Cotter
7. C. Hill

In Surgery

1. Stefan H. E. Kaufmann
2. B. B. Finlay
3. G. McFadden
4. Paul D. Cotter
5. C. Hill
6. M. Gengenbacher

Citations in the corpus

• Transcriptional Adaptation of Mycobacterium tuberculosis within Macrophages (2003, background): “MTB may achieve this effect by controlling phagosomal development, preventing its fusion with the lysosome, and reducing maximal acidification of phagosomal contents (5).”
• Mycobacterium tuberculosis: Success through dormancy (2012, background): “However, Mtb manages to escape eradication by macrophages and survives within these cells (Armstrong & Hart, 1975; Kaufmann, 2001; Russell, 2001).”
• Immune-responsive gene 1 protein links metabolism to immunity by catalyzing itaconic acid production (2013, background): “Because the glyoxylate shunt is exclusively found in prokaryotes, lower eukaryotes, and plants, it represents a unique target for drug development (37).”
• DC-SIGN Is the Major Mycobacterium tuberculosis Receptor on Human Dendritic Cells (2003, background): “Journal of Experimental Medicine, Rockefeller University Press, 2003, 197 (1), pp.”
• Deletion of RD1 from Mycobacterium tuberculosis mimics bacille Calmette-Guérin attenuation. (2003, neutral): “The macrophage is the primary cell type to interact directly with MTB in vivo [17], and growth in cultured macrophages has been used to assess virulence of different mycobacterial strains [18, 19].”
• M. tuberculosis and M. leprae translocate from the phagolysosome to the cytosol in myeloid cells. (2007, background): “The prevailing paradigm has remained that M. tuberculosis reside in the endocytic system (Orme, 2004; Vergne et al., 2004; Russell et al., 2002; Kang et al., 2005; Russell, 2001; Pizarro-Cerda and Cossart, 2006).”
• Anti-immunology: evasion of the host immune system by bacterial and viral pathogens. (2006, background): “M. tuberculosis, which is probably the most successful intracellular human pathogen , has many surface glycolipids and carbohydrates that prevent phagosome acidification and alter phagosomes (Russell, 2001).”
• Surviving the Acid Test: Responses of Gram-Positive Bacteria to Low pH (2003, background): “Pathogenic mycobacteria alter the phagosomal pH by excluding vacuolar ATPase from the phagosome membrane or inhibiting phagosome-lysosome fusion (197).”