A new population of myeloid-derived suppressor cells in hepatocellular carcinoma patients induces CD4(+)CD25(+)Foxp3(+) T cells.
By Bastian Hoechst, Lars A. Ormandy, M. Ballmaier, F. Lehner, C. Krüger, M. Manns, T. Greten, F. Korangy. Gastroenterology, 2008
822 citations recorded by the source corpus.
Subfields: Education, Information Systems
View Paper: https://www.semanticscholar.org/paper/d736d4027c6e6b1aa1b3be982950d9514dc2b7d2
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Citations in the corpus
- The Role of Cancer-Associated Fibroblasts and Fibrosis in Liver Cancer (2016, background): “Several lines of evidence suggest that HSCs reduce tumor immunosurveillance through paracrine signals to surrounding immune cells: (a) Cotransplantation studies demonstrated that HSCs inhibit lymphocyte infiltration in tumors and the spleens of tumor-bearing mice, induce apoptosis of infiltrating mo”
- The role of myeloid cells in the promotion of tumour angiogenesis (2008, background): “The presence of MDSC has been described in the peripheral blood and tumours of patients with various forms of cance”
- Advances in immunotherapy for hepatocellular carcinoma (2021, background): “I.M. receives funding from MINECO SAF2014-52361-R and SAF 2017- 83267-C2-1R; Worldwide Cancer Research Grant (15-1146); the Asociacion Española Contra el Cancer (AECC) Foundation under grant GCB15152947MELE; and the European Union’s Horizon 2020 Program (grant agreement no. 635122 PROCROP).”
- Myeloid-Derived Suppressor Cells: Linking Inflammation and Cancer (2009, background): “A new population of MDSC was recently identified in melanoma and hepatocarcinoma patients that is CD14 HLA-DR , suggesting that, similarly as tumors in mice, different human tumors are likely to induce different subtypes of MDSC (30, 49).”
- Regulatory T cells in tumor microenvironment: new mechanisms, potential therapeutic strategies and future prospects (2020, background): “Hirata A, Hashimoto H, Shibasaki C, Narumi K, Aoki K. Intratumoral IFN-alpha gene delivery reduces tumor-infiltrating regulatory T cells through the downregulation of tumor CCL17 expression.”