Moderate exercise is an antioxidant: upregulation of antioxidant genes by training.
By M. Gómez-Cabrera, Elena Doménech, J. Viña. Free Radical Biology & Medicine, 2008
1,035 citations recorded by the source corpus.
Subfields: Nutrition and Dietetics, Physiology, Rehabilitation
View Paper: https://www.semanticscholar.org/paper/ca0a3dde48fd4879cdd6f04c31fd122c335dc475
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Citations in the corpus
- Oxidative Stress, Prooxidants, and Antioxidants: The Interplay (2014, background): “Mild physical activity increases nuclear factor-kappa B (NF-κB) activity in the muscle of rats as well as the gene expression for manganese superoxide dismutase (MnSOD) and endothelial nitric oxide synthase (eNOS) [33].”
- Acute exercise and oxidative stress: a 30 year history (2009, background): “However, whether this exerciseinduced increase in RONS exerts detrimental effects on long term physiological function remains a topic of debate, as an ever increasing body of evidence in the area suggests that biologically-derived RONS act in a hormetic manner [9,312,313].”
- Inflammation and aging: signaling pathways and intervention therapies (2023, background): “Exercise is an efficient strategy for delaying aging(330) through various mechanisms, such as DNA damage(331) and oxidative stress.(332) Recent study found that middle-aged marathon/ triathletes had higher telomerase activity and longer telomere length in circulating white blood cells compared to th”
- Exercise is the real polypill. (2013, background): “In fact, muscle-derived ROS occurring during prolonged inactivity contribute to disuse muscle atrophy (382, 383), whereas the same stimulus coming from working fibers is required for training adaptations to occur (149, 150, 168, 404).”