Erratum: Liver fibrosis (Journal of Clinical Investigation (2005) 115 (209-218) DOI:10.1172/JCI200524282)

By Ramón Bataller, D. A. Brenner. 2005

1,195 citations recorded by the source corpus.

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

Non-cell-autonomous tumor suppression by p53 (2013, positive): “…an ongoing cycle of cell death, HSC proliferation, inflammation, and fibrosis eventually leads to organ failure through cirrhosis, which is the strongest risk factor for the emergence of hepatocellular carcinoma (HCC)—the third leading cause of cancer deaths worldwide (Bataller and Brenner, 2005).”
Recent advances in 2D and 3D in vitro systems using primary hepatocytes, alternative hepatocyte sources and non-parenchymal liver cells and their use in investigating mechanisms of hepatotoxicity, cell signaling and ADME (2013, positive): “Their role in pathology is mostly addressed in conditions of chronic liver damage, where their main function is the deposition of ECM, which limits the progression of injury and favors tissue regeneration (Bataller and Brenner 2005; Radaeva et al. 2006).”