VEGF inhibition and renal thrombotic microangiopathy.
By D. Nochy, C. Lefaucheur, G. Hill. New England Journal of Medicine, 2008
876 citations recorded by the source corpus.
View Paper: https://www.semanticscholar.org/paper/d557f83cc12583c264d058931f50a5d2d4a77c26
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Citations in the corpus
- 2016 ESC Position Paper on cancer treatments and cardiovascular toxicity developed under the auspices of the ESC Committee for Practice Guidelines: The Task Force for cancer treatments and cardiovascular toxicity of the European Society of Cardiology (ESC). (2016, background): “9, respectively, under bevacizumab, sorafenib and sunitinib.(163,164) A summary of the incidence of hypertension reported in patients with cancer taking these drugs can be found in the appendix at the end of this document.”
- Molecular mechanisms of diabetic kidney disease. (2014, unknown): “For example, both increased and decreased VEGF expression causes albuminuria and glomerular changes (57, 58).”
- Autophagy influences glomerular disease susceptibility and maintains podocyte homeostasis in aging mice. (2010, methodology): “Podo- cin-rtTA ;tetO-Cre mice were provided by S. Quaggin (Samuel Lunenfeld Research Institute, Mount Sinai Hospital, University of Toronto, Canada) (49).”
- Organotypic vasculature: From descriptive heterogeneity to functional pathophysiology (2017, background): “VEGF blockademay cause proteinuria and thrombotic microangiopathy (126).”