Diabetic Nephropathy: Mechanisms of Renal Disease Progression
By Y. Kanwar, J. Wada, Lin Sun, P. Xie, E. Wallner, Sheldon Chen, S. Chugh, Farhad R. Danesh. Experimental biology and medicine, 2008
619 citations recorded by the source corpus.
Subfields: Clinical Biochemistry, Nephrology, Neurology
View Paper: https://www.semanticscholar.org/paper/7938e38e782271e09fdc120b023a3c2b83add3fb
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Citations in the corpus
- The Diabetes Mellitus–Atherosclerosis Connection: The Role of Lipid and Glucose Metabolism and Chronic Inflammation (2020, background): “Both persistent and temporal hyperglycemic exposure were shown to influence several significant cellular signaling pathways, including PKC activation and oxidative stress, described above, and transforming growth factor (TGF)-β-SMAD-MAPK signaling [73,74].”
- Relationship between oxidative stress and inflammatory cytokines in diabetic nephropathy. (2012, background): “Increased oxidative stress in diabetes likely contributes to the pathogenesis of diabetic nephropathy and its progression to end-stage renal disease [18–20].”