Inflammation and cancer: An ancient link with novel potentials

By S. Perwez Hussain, C. Harris. International Journal of Cancer, 2007

1,116 citations recorded by the source corpus.

Subfields: Cancer Research, Immunology

View Paper: https://www.semanticscholar.org/paper/d4a3e75f5dd45e8f946104f762daa9d7ad918bc8

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cancer Research

1. Bharat B. Aggarwal
2. Subash C. Gupta
3. Y. Surh
4. J. Kundu
5. F. Greten
6. Simone Reuter
7. H. Hermeking
8. M. Karin

In Immunology

1. Bharat B. Aggarwal
2. Subash C. Gupta
3. Y. Lin
4. Xia Wang
5. F. Greten
6. Simone Reuter
7. M. Karin
8. K. Harikumar

Citations in the corpus

• Upsides and Downsides of Reactive Oxygen Species for Cancer: The Roles of Reactive Oxygen Species in Tumorigenesis, Prevention, and Therapy (2012, neutral): “Induction of COX-2, inflamma-tory cytokines (TNF-a , interleukin [IL]-1, IL-6), chemokines (IL-8, CXCR4), and pro-inflammatory transcription factors ( e.g. , NF-j B)—all well-known mediators of inflammation and tumorigenesis—is regulated by ROS (131, 222).”
• Luteolin, a flavonoid with potential for cancer prevention and therapy. (2008, background): “However, chronic inflammation may result in harmful diseases such as arthritis, chronic obstructive pulmonary disease, and cancer [48-50].”
• Role of Antioxidants and Natural Products in Inflammation (2016, background): “Initiation of cyclooxygenase-2 (COX-2), inducibility of nitric oxide synthase (iNOS), and high expression of inflammatory cytokines, including tumor necrosis factor-α (TNF-α), interleukin-1β (IL-1β), IL-6, and chemokines (CXC chemokine receptor 4), in addition to changes in the expression of specifi”
• Oxidative stress, inflammation, and cancer: how are they linked? (2010, background): “specific microRNAs have also been reported to play a role in oxidative stress-induced inflammation [69].”
• Inflammation: gearing the journey to cancer. (2008, background): “chronic infection and inflammation [3].”
• Chronic Inflammation and Cytokines in the Tumor Microenvironment (2014, background): “In the last decades, Virchow’s postulation has been supported by abundant evidence that various cancers are triggered by infection and chronic inflammatory disease [3].”
• IL-6R/STAT3/miR-34a feedback loop promotes EMT-mediated colorectal cancer invasion and metastasis. (2014, background): “About 25% of cancers appear due to chronic infection or other types of chronic inflammation (7).”