Lead neurotoxicity in children: basic mechanisms and clinical correlates.
By T. Lidsky, J. Schneider. Brain : a journal of neurology, 2003
1,124 citations recorded by the source corpus.
Subfields: Health, Toxicology and Mutagenesis, Nutrition and Dietetics, Speech and Hearing
View Paper: https://www.semanticscholar.org/paper/d3f5036141463d12fa2fefc251d743b65406d389
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Citations in the corpus
- Pb Neurotoxicity: Neuropsychological Effects of Lead Toxicity (2014, neutral): “Numerous studies of children have shown that lead exposure reduces overall cognitive functioning in children, both cross-sectionally and longitudinally, but most such studies examine omnibus measures of intellectual functioning rather than domainspecific effects [35, 42–44].”
- Effects of air pollution on human health and practical measures for prevention in Iran (2016, neutral): “Mental retardation, learning disabilities, impairment of memory, hyperactivity, and antisocial behaviors are of adverse effects of Pb in childhood.[57,58]”
- Low-Level Environmental Lead Exposure and Children’s Intellectual Function: An International Pooled Analysis (2005, background): “…failure, delinquent behavior, hearing loss, tooth decay, spontaneous abortions, renal disease, and cardiovascular disease (Borja-Aburto et al. 1999; Dietrich et al. 2001; Factor-Litvak et al. 1999; Lin et al. 2003; Moss et al. 1999; Nash et al. 2003; Needleman et al. 2002; Schwartz and Otto 1991).”
- An Overview of Catastrophic AI Risks (2023, background): “For example, lead was widely used in products like paint and gasoline until its neurotoxic effects came to light [78].”
- Practice parameter for the assessment and treatment of children and adolescents with attention-deficit/hyperactivity disorder. (2007, background): “Exposure to lead, either prenatally or during development, is associated with a number of neurocognitive impairments, including ADHD (Lidsky and Schneider, 2003).”
- Human pluripotent stem cell-derived neural constructs for predicting neural toxicity (2015, background): “…to assess developmental neurotoxicity in vitro, as the underlying mechanisms may include selective cell death, delayed or aberrant differentiation, suppressed neurotransmission, disruption of the blood – brain barrier, or modulation of inflammatory signals by glial or microglial cells (16 – 20).”