The role of cerebrospinal fluid hypocretin measurement in the diagnosis of narcolepsy and other hypersomnias.

By E. Mignot, G. Lammers, B. Ripley, M. Okun, S. Nevsimalova, S. Overeem, J. Vanková, J. Black, J. Harsh, C. Bassetti and 2 more. Archives of Neurology, 2002

1,089 citations recorded by the source corpus.

Subfields: Cognitive Neuroscience, Experimental and Cognitive Psychology

View Paper: https://www.semanticscholar.org/paper/d3709a92a59fcea50a20e16ff04118cdac29e235

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In Cognitive Neuroscience

1. T. Sakurai

In Experimental and Cognitive Psychology

1. T. Sakurai

Citations in the corpus

• Clinical analysis of anti-Ma2-associated encephalitis. (2004, background): “The EDS was likely due to hypothalamic dysfunction because the hypocretin levels were very low or undetectable in the CSF of patients with EDS but were normal in patients without EDS (Mignot et al., 2002).”
• The neural circuit of orexin (hypocretin): maintaining sleep and wakefulness (2007, background): “Approximately 90% of patients with narcolepsy, especially those with narcolepsy–cataplexy, show decreased orexin A levels in the cerebrospinal flui”