Regulation of the Mycobacterium tuberculosis hypoxic response gene encoding α-crystallin

By David R. Sherman, M. Voskuil, D. Schnappinger, R. Liao, M. Harrell, G. Schoolnik. Proceedings of the National Academy of Sciences of the United States of America, 2001

700 citations recorded by the source corpus.

Subfields: Immunology, Infectious Diseases

View Paper: https://www.semanticscholar.org/paper/d2e1bf4efefd25a88b0edf5aaa71d5ad8f6e33ec

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Citations in the corpus

• Rv3133c/dosR is a transcription factor that mediates the hypoxic response of Mycobacterium tuberculosis (2003, methodology): “© 2003 Blackwell Publishing Ltd, Molecular Microbiology, 48, 833–843 information necessary to direct the hypoxic induction of acr (Yuan et al., 1998; Sherman et al., 2001).”
• Deletion of RD1 from Mycobacterium tuberculosis mimics bacille Calmette-Guérin attenuation. (2003, unknown): “Electroporation into mycobacteria, initial selection on 7H10 plates with hygromycin, and secondary selection on 7H10 plates containing 10% sucrose were performed as described elsewhere [8, 14].”
• Foamy Macrophages from Tuberculous Patients' Granulomas Constitute a Nutrient-Rich Reservoir for M. tuberculosis Persistence (2008, methodology): “For this purpose, we analyzed the expression of a series of genes known to be up-regulated when bacilli are in a persistent non-replicating state [23,24].”