Neuregulin-1 type III determines the ensheathment fate of axons.

By C. Taveggia, G. Zanazzi, Ashley Petrylak, H. Yano, J. Rosenbluth, S. Einheber, Xiaorong Xu, R. Esper, J. Loeb, P. Shrager and 4 more. Neuron, 2005

735 citations recorded by the source corpus.

Subfields: Cellular and Molecular Neuroscience, Immunology and Allergy, Oncology

View Paper: https://www.semanticscholar.org/paper/d285b14bf7dd879e6c57b91e2b4d5684f29a129b

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In Cellular and Molecular Neuroscience

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In Immunology and Allergy

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In Oncology

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Citations in the corpus

• Bace1 modulates myelination in the central and peripheral nervous system (2006, background): “and as a modulator of myelin sheath thickness in both the CNS and PN”
• Myelination and support of axonal integrity by glia (2010, background): “Although the mechanism that led to the evolution of myelin in such jawless fish may never be known, it is intriguing that overexpression of a single axonal growth factor turns non-myelinating into myelinating Schwann cell”
• Axon-glial signaling and the glial support of axon function. (2008, background): “Following expansion of the Schwann cell pool in the developing peripheral nerve, axonbound NRG1 type III is required for the differentiation of the myelinating Schwann cell phenotype (Leimeroth et al. 2002, Taveggia et al. 2005).”