Glucose-induced reactive oxygen species cause apoptosis of podocytes and podocyte depletion at the onset of diabetic nephropathy.

By K. Susztak, Amanda C. Raff, M. Schiffer, Erwin P. Bottinger. Diabetes, 2006

1,118 citations recorded by the source corpus.

Subfields: Clinical Biochemistry, Nephrology

View Paper: https://www.semanticscholar.org/paper/d226318e8f282e65147d390849bcc74e274cca22

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

1. J. Stern
2. Karolina Woroniecka
3. J. Rutkowski
4. P. Scherer
5. Ae Seo Deok Park
6. D. Mohtat
7. David B. Thomas
8. J. Pullman

Citations in the corpus

• Transcriptome Analysis of Human Diabetic Kidney Disease (2011, background): “Decreased podocyte number has been proposed to play a key role in DKD (31).”
• Adiponectin, Leptin, and Fatty Acids in the Maintenance of Metabolic Homeostasis Through Adipose Tissue Crosstalk (2016, background): “A similar outcome occurs in glomerular podocytes, wherein hyperglycemia incudes ROSmediated apoptosis, a first step in the progression of diabetic nephropathy (Susztak et al., 2006).”