Annexin A1 and glucocorticoids as effectors of the resolution of inflammation

By M. Perretti, F. D’Acquisto. Nature reviews. Immunology, 2009

919 citations recorded by the source corpus.

Subfields: Immunology, Molecular Biology, Oncology

View Paper: https://www.semanticscholar.org/paper/cd8b789e0968ac00a6ee6806af6a82b5b67e7045

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Immunology

1. C. Serhan
2. A. Lichtman
3. Oliver Soehnlein
4. N. Petasis
5. I. Tabas
6. L. Lindbom
7. Chia Nan
8. Nikolaos G. Frangogiannis

In Molecular Biology

1. A. Lichtman
2. C. Serhan
3. I. Tabas
4. Oliver Soehnlein
5. L. Lindbom
6. C. A. Dinarello
7. J. Malhotra
8. R. Kaufman

In Oncology

1. I. Tabas
2. A. Lichtman

Citations in the corpus

• Monocyte-Macrophages and T Cells in Atherosclerosis (2017, positive): “By activating specific cell-surface receptors, these mediators block inflammatory cell influx and promote their egress; clear pathogens, cellular debris, inflammatory cytokines, and apoptotic cells (efferocytosis); and repair tissue damage (Serhan, 2010; Perretti and D’Acquisto, 2009).”
• Phagocyte partnership during the onset and resolution of inflammation (2010, background): “Following cell activation, annexin A1 is rapidly discharge”
• Resolvin D1 binds human phagocytes with evidence for proresolving receptors (2010, methodology): “Competition binding was performed with RvD1, LXA4, and the annexin 1-derived peptide Ac2-12, an antiinflammatory peptide ligand that binds to ALX (17).”