Bacterial Invasion: The Paradigms of Enteroinvasive Pathogens

By P. Cossart, P. Sansonetti. Science, 2004

987 citations recorded by the source corpus.

View Paper: https://www.semanticscholar.org/paper/cd305d8729bc9794a3b58304512b2c3bfc19de52

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Citations in the corpus

The Legionella Kinase LegK2 Targets the ARP2/3 Complex To Inhibit Actin Nucleation on Phagosomes and Allow Bacterial Evasion of the Late Endocytic Pathway (2015, positive): “Actin cytoskeleton remodeling is known to be targeted by numerous extra- and intracellular bacteria in order to (i) trigger bacterial uptake by nonphagocytic cells (42), (ii) move within the host cell cytosol and spread from cell to cell (43), or (iii) decorate the phagocytic vacuole with a polymeri”
War and peace at mucosal surfaces (2004, background): “Rupture of the intestinal barrier by Gram-negative entero-invasive pathogens can lead to three main outcomes, depending on the intrinsic virulent properties of the pathoge”
Stochastic mechano-chemical kinetics of molecular motors: A multidisciplinary enterprise from a physicist’s perspective (2012, background): “comets” [983, 984, 985, 986, 987, 988, 989, 990, 991, 992] (for an historical account of its discovery, see ref[993]).”
Invasive and Adherent Bacterial Pathogens Co-Opt Host Clathrin for Infection (2007, positive): “Inhibiting actin rearrangements using the fungal metabolite cytochalasin D or other strategies such as depletion of dynamin, cortactin, or CD2AP by siRNA prevent bacterial internalization ( Cossart and Sansonetti, 2004; Veiga and Cossart, 2005).”
Intestinal microbiota and metabolites—Implications for broiler chicken health and performance1 (2013, background): “After normal host-cell processes have been weakened, these systems enable the pathogen to penetrate and cross the epithelial barrier [58].”
Culture independent analysis of ileal mucosa reveals a selective increase in invasive Escherichia coli of novel phylogeny relative to depletion of Clostridiales in Crohn's disease involving the ileum (2007, background): “Bacterial virulence typically is regulated by genes that promote invasion or replication of organisms in target cells (Cossart and Sansonetti, 2004).”
Molecular Pathogenesis of Shigella spp.: Controlling Host Cell Signaling, Invasion, and Death by Type III Secretion (2008, background): “flexneri depends on complex rearrangements of membranes and the actin cytoskeleton, which are mediated by the controlled temporal and spatial actions of a multitude of bacterial and host cell factors (44, 310) (Fig.”
Septins: the fourth component of the cytoskeleton (2012, background): “Functions of septins in infection The study of host–microorganism interactions has been instrumental in investigating the functions of cytoskeletal components (reviewed in Ref”
Bacterial adhesion and entry into host cells. (2006, neutral): “Both pathogens trigger important cytoskeletal changes at the host-cell plasma membrane to promote invasion (Cossart and Sansonetti, 2004).”
Inflammasome adaptors and sensors: intracellular regulators of infection and inflammation (2007, background): “Alternatively, SipB and IpaB have also been shown to intercalate with the membrane lipid rafts of host cells and set the stage for actin rearrangements through deactivation of RhoA GTPase, leading to RAC1 activatio”