Maternal cigarette smoking, metabolic gene polymorphism, and infant birth weight.
By Xiaobin Wang, B. Zuckerman, C. Pearson, G. Kaufman, Changzhong Chen, Guoying Wang, Tianhua Niu, P. Wise, H. Bauchner, Xiping Xu. Journal of the American Medical Association (JAMA), 2002
612 citations recorded by the source corpus.
Subfields: Cancer Research, Oncology, Physiology
View Paper: https://www.semanticscholar.org/paper/cca6788fa32e38838d23d69004840a99b20e20a5
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Citations in the corpus
- The epidemiology of smoking during pregnancy: smoking prevalence, maternal characteristics, and pregnancy outcomes. (2004, background): “Because only a minority of smokers have growth-restricted infants, Wang et al. (2002) investigated whether genetic factors might modify the smoking-related risk of reduced birth weight.”
- Polymorphism of human cytochrome P450 enzymes and its clinical impact (2009, background): “When both CYP1A1 and GSTT1 genotypes were considered, the greatest reduction in birthweight among smoking mothers with the CYP1A1 Aa/aa and GSTT1-deficient genotypes (Wang et al., 2002b).”
- Gene-environment interplay and psychopathology: multiple varieties but real effects. (2006, background): “In a study of low infant birth weight, women who smoked tobacco during pregnancy gave birth to underweight infants, or did not, depending on their genotype with respect to two polymorphic metabolic genes, CYP1A1 and GSTT1 (Wang et al., 2002).”
- Epidemiology and causes of preterm birth (2008, background): “138 The data provide evidence for gene-environment interactions in spontaneous preterm birth.”