Enhanced release of superoxide from polymorphonuclear neutrophils in obstructive sleep apnea. Impact of continuous positive airway pressure therapy.
By R. Schulz, S. Mahmoudi, K. Hattar, U. Sibelius, H. Olschewski, K. Mayer, W. Seeger, F. Grimminger. American Journal of Respiratory and Critical Care Medicine, 2000
647 citations recorded by the source corpus.
Subfields: Neurology, Physiology, Pulmonary and Respiratory Medicine
View Paper: https://www.semanticscholar.org/paper/cb200c3090585216ad433027d25d06737c9c190e
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In Physiology
Citations in the corpus
- Reactive Oxygen Species in Inflammation and Tissue Injury (2014, neutral): “In addition, in patients suffering from obstructive sleep apnea, an increased expression of adhesion molecules CD15 (a counterreceptor for selectins on ECs), CD11c (counter-receptor for ICAM-1 on ECs) on monocytes was correlated to increased ROS generation (105, 381).”
- Endothelial function in obstructive sleep apnea and response to treatment. (2004, background): “Other mechanisms for impaired nitric oxide–mediated endothelial function, including increased oxidant load, enhanced oxidation of low density lipoprotein, insulin resistance, and increased sympathetic activity (46, 47), have been demonstrated to be present in OSA (10, 33, 48 , 49).”
- Obstructive sleep apnea: implications for cardiac and vascular disease. (2003, background): “Prevention of OSA by CPAP reduces production of superoxide.(54)”