Obstructive sleep apnea: implications for cardiac and vascular disease.

By A. Shamsuzzaman, B. J. Gersh, Virend K. Somers. Journal of the American Medical Association (JAMA), 2003

1,314 citations recorded by the source corpus.

Subfields: Experimental and Cognitive Psychology, Physiology, Pulmonary and Respiratory Medicine

View Paper: https://www.semanticscholar.org/paper/3ff7a2b2e1fced6e689a49b04bd30eda803f1e1f

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In Experimental and Cognitive Psychology

In Physiology

In Pulmonary and Respiratory Medicine

Citations in the corpus

Sleep apnea and cardiovascular disease: an American Heart Association/American College of Cardiology Foundation Scientific Statement from the American Heart Association Council for High Blood Pressure Research Professional Education Committee, Council on Clinical Cardiology, Stroke Council, and Coun (2008, background): “OSA could potentially contribute to the progression of heart failure through several pathological mechanisms: (1) by eliciting greater sympathetic outflow to the heart, kidney, and resistance vessels during wakefulness and sleep; (2) by increasing left ventricular afterload both acutely and chronica”
Risk factors for obstructive sleep apnea in adults. (2004, background): “Obstructive sleep apnea is associated with diabetes, hypertension, coronary artery disease, myocardial infarction, congestive heart failure, and stroke.(1,3,29) The associations may be due in part to risk factors common to all these conditions; they may also reflect a role of OSA in the etiology of ”
European Guidelines on Cardiovascular Disease Prevention in Clinical Practice (Version 2012) (2012, neutral): “Repetitive bursts of sympathetic activity, surges of blood pressure, and oxidative stress brought on by pain and episodic hypoxaemia associated with increased levels of mediators of inflammation are thought to promote endothelial dysfunction and atherosclerosis.(176) OSA has been associated with a 7”
Association of sleep-disordered breathing and the occurrence of stroke. (2005, background): “The authors of recent reviews of this subject came to the conclusion that, whereas there is evidence favoring SDB as a modifiable risk factor for stroke, this evidence has not established a relationship of SDB with stroke that is independent of known risk factors, and that prospective studies are ne”