Postconditioning: A Form of “Modified Reperfusion” Protects the Myocardium by Activating the Phosphatidylinositol 3-Kinase–Akt Pathway

By A. Tsang, D. Hausenloy, M. Mocanu, D. Yellon. Circulation Research, 2004

710 citations recorded by the source corpus.

Subfields: Cardiology and Cardiovascular Medicine, Emergency Medicine, Pathology and Forensic Medicine

View Paper: https://www.semanticscholar.org/paper/c9bd1b9b0479bcd3441ef3a8365367b82629b0fc

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cardiology and Cardiovascular Medicine

1. E. Murphy
2. C. Steenbergen

In Emergency Medicine

1. E. Murphy
2. C. Steenbergen

In Pathology and Forensic Medicine

1. E. Murphy
2. C. Steenbergen

Citations in the corpus

• Mechanisms underlying acute protection from cardiac ischemia-reperfusion injury. (2008, background): “2) PI3K— Tsang et al (259) demonstrated in rat heart that postconditioning activates Akt, eNOS, and p70S6K, and that activation is blocked by inhibitors of PI3K.”