The Fetal and Childhood Growth of Persons Who Develop Type 2 Diabetes
By Tom Forsn, J. Eriksson, J. Tuomilehto, A. Reunanen, C. Osmond, D. Barker. Annals of Internal Medicine, 2000
741 citations recorded by the source corpus.
Subfields: Endocrinology, Diabetes and Metabolism, Obstetrics and Gynecology, Pediatrics, Perinatology and Child Health
View Paper: https://www.semanticscholar.org/paper/c61d6adf94da35fa7b1ebd4332607ece26b12cff
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Citations in the corpus
- A life course approach to chronic disease epidemiology. (2005, background): “This seems to be the case for the associations of birth weight with some chronic diseases in which associations are stronger (or only evident) among those who become obese during adolescence or adulthood (61, 69, 71).”
- Birth weight and subsequent risk of type 2 diabetes: a meta-analysis. (2007, background): “, 2000 (19) Finland Cohort 1924–1933 64–73 0.”
- Social Determinants of Risk and Outcomes for Cardiovascular Disease: A Scientific Statement From the American Heart Association (2015, background): “Insulin resistance and type 2 diabetes mellitus also appear to be more prevalent in adults with intrauterine growth retardation, and the same pattern exists.(324,325) In a population-based study from India, those individuals who developed glucose intolerance or type 2 diabetes mellitus had significa”
- Birth weight and risk of type 2 diabetes: a systematic review. (2008, background): “Information on physiciandiagnosed diabetes by individual recall wasused in21studies§and information on physician-diagnosed diabetes from medicalregistersorotherroutinesources by 3 studies.(15,16,19) In 14 studies, the diagnosis of diabetes was based on a combinationof recallofphysician-diagnosed dia”
- A life course approach to diet, nutrition and the prevention of chronic diseases (2004, background): “Several studies have demonstrated an interaction between rapid catch-up growth in weight and IUGR in increased risk of disease, e.g. in Finland where the highest risk of CHD or metabolic syndrome, for both men and women, was found to be associated with this sequence 22,27,66 .”
- Childhood obesity as a predictor of morbidity in adulthood: a systematic review and meta‐analysis (2016, background): “type II diabetes (two studies) (37,47), coronary heart disease (one study) (48), hypertension (six studies) (32–35,37,49), the metabolic syndrome (five studies) (18,20,33,34,50), hypercholesterolaemia (one study) (51) and renal cell carcinoma (one study) (52).”
- The developmental origins of chronic adult disease (2004, background): “There are a number of processes by which, in humans, undernutrition and small size at birth followed by rapid childhood growth could lead to cardiovascular disease and type 2 diabetes in later life (11, 14).”
- Fetal Origins of Adult Disease (2011, neutral): “Infants born SGA are at ncreased risk for developing obesity, T2DM, CAD, ypertension, kidney disease, premature pubarche, and polycystic ovarian syndrome, dyslipidemia, short stature, and osteoporosis.(1-4,13,32,37,45,46,49,50) Many of these phenotypic changes are known to be secondary to or associa”
- The Developmental Origins of Adult Disease (2004, background): “There are a number of processes by which, in humans, undernutrition and small size at birth followed by rapid childhood growth could lead to cardiovascular disease and type 2 diabetes in later life [10,13].”