The Developmental Origins of Adult Disease
By D. Barker. Journal of the American College of Nutrition, 2004
1,140 citations recorded by the source corpus.
Subfields: Obstetrics and Gynecology, Pediatrics, Perinatology and Child Health, Public Health, Environmental and Occupational Health
View Paper: https://www.semanticscholar.org/paper/98e4e15476070424faf7517c181782a420c91dc1
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Obstetrics and Gynecology
In Pediatrics, Perinatology and Child Health
- Dana C. Dolinoy
- Karilyn E. Sant
- Olivia S. Anderson
- Elizabeth C. Cottrell
- R. Jirtle
- Robert A. Waterland
- C. Gravlee
- J. Seckl
In Public Health, Environmental and Occupational Health
- E. Diamanti-Kandarakis
- Elizabeth C. Cottrell
- C. Gravlee
- J. Seckl
- Dana C. Dolinoy
- Karilyn E. Sant
- Olivia S. Anderson
Citations in the corpus
- The long-term behavioural consequences of prenatal stress. (2008, neutral): “COR and other hormones could then have produced alterations in its structure and function depending on the hormone and the amount and time of elevation in relation to the appearance of particular neural and endocrine systems and of steroid and catecholamine receptors.”
- Maternal Genistein Alters Coat Color and Protects Avy Mouse Offspring from Obesity by Modifying the Fetal Epigenome (2006, background): “Specifically, the developmental origins hypothesis postulates that nutrition and other environmental factors during prenatal and early postnatal development influence developmental plasticity and alter susceptibility to adult cardiovascular disease, type 2 diabetes, and obesity (Barker 1997, 2004).”
- Nutrition and epigenetics: An interplay of dietary methyl donors, one-carbon metabolism, and DNA methylation (2012, positive): “The study of nutri-epigenomics is particularly timely in the context of the developmental origins of health and disease (DOHaD) hypothesis [18], which posits that increased susceptibility to disease following early life experiences is shaped by epigenetic modifications such as DNA methylation and ch”
- How race becomes biology: embodiment of social inequality. (2009, background): “…al., 2008), but there is also a lingering effect into adulthood, as adult chronic diseases like heart disease and diabetes can be traced in part to prenatal and early life conditions (Barker, 2004; Adair and Dahly, 2005; Cruickshank et al., 2005; Pollitt et al., 2005; Junien and Nathanielsz, 2007).”
- Prenatal Stress, Glucocorticoids and the Programming of Adult Disease (2009, background): “Furthermore, these associations are continuous across a wide range of normal birth weights, and act largely independently of other lifestyle factors such as socioeconomic status and current obesity (Barker, 2004).”
- Endocrine-disrupting chemicals: an Endocrine Society scientific statement. (2009, background): “In fact, the field of endocrine disruption has embraced the terminology “the fetal basis of adult disease” (8) to describe observations that the environ ment of a developing organism, which includes the maternal environment (eutherian mammals), the egg (other vertebrates), and the external environm”