Comparing signaling mechanisms engaged in pattern-triggered and effector-triggered immunity.

By Kenichi Tsuda, Fumiaki Katagiri. Current opinion in plant biology, 2010

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Of PAMPs and Effectors: The Blurred PTI-ETI Dichotomy[OA] (2011, background): “…discriminate two phases of plant immunity: PTI triggered by PAMPs and ETI triggered by effectors, with the paradigm that activated immune responses in ETI occur quicker and are more prolonged and more robust than those in PTI (Tao et al., 2003; Jones and Dangl, 2006; Tsuda and Katagiri, 2010).”
Signaling in Plant-Microbe Interactions (2013, background): “Generally, PAMPs induce transient/low amplitude of ROS production, however R-protein directed pathogen effector recognition leads to sustained and high amplitude of ROS production (Torres et al. 2006; Tsuda and Katagiri 2010).”
Comprehending crystalline β-carotene accumulation by comparing engineered cell models and the natural carotenoid-rich system of citrus (2011, neutral): “Pathogens cannot sneak in: upon contact with invaders, plant cells use the first branch of their innate immune system by perceiving conserved microbial structures and peptides with the help of plasma membrane-localized PRRs (Fig. 1; Zipfel, 2009; Tsuda and Katagiri, 2010).”