Acute axonal injury in multiple sclerosis. Correlation with demyelination and inflammation.
By A. Bitsch, J. Schuchardt, S. Bunkowski, T. Kuhlmann, Wolfgang Brück. Brain : a journal of neurology, 2000
961 citations recorded by the source corpus.
Subfields: Neurology, Pathology and Forensic Medicine, Rheumatology
View Paper: https://www.semanticscholar.org/paper/c49c8eb8b12c3ef839146e94e8940454c6786605
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Citations in the corpus
- New concepts in the immunopathogenesis of multiple sclerosis (2002, critical): “The axonal damage, however, can also be observed in the absence of inflammation and demyelination, questioning whether immune-independent mechanisms are also involved in the neurodegenerative damage in M”
- Acute axonal damage in multiple sclerosis is most extensive in early disease stages and decreases over time. (2002, background): “A variety of different histopathological studies exist that investigated acute axonal damage in multiple sclerosis (Ferguson et al., 1997; Trapp et al., 1998; Bitsch et al., 2000; Kornek et al., 2000).”
- The neurobiology of multiple sclerosis: genes, inflammation, and neurodegeneration. (2006, background): “…highresolution histopathological studies reveal abundant transected and dystrophic axons in sites of active inflammation and demyelination, and confirm that partial or total axonal transection begins early in the disease process (Bitsch et al., 2000; Ferguson et al., 1997; Trapp et al., 1998).”
- Chemokine receptors: multifaceted therapeutic targets (2002, background): “In the case of the truncated CCR2 antagonist (9-68)-CCL2, the efficacy of blockade of CCR2 has been shown in the spontaneous model of arthritis in”
- Radial Diffusivity Predicts Demyelination in ex-vivo Multiple Sclerosis Spinal Cords (2011, neutral): “A complication in examining ex vivo spinal cords includes the complex interplay of these pathologies that can be present in concert or independently to some extent (Bitsch et al., 2000; DeLuca et al., 2006).”