Biology of gastrointestinal stromal tumors.
By Christopher Corless, J. Fletcher, M. Heinrich. Journal of Clinical Oncology, 2004
1,198 citations recorded by the source corpus.
Subfields: Gastroenterology, Neurology, Pulmonary and Respiratory Medicine
View Paper: https://www.semanticscholar.org/paper/c15abb52fa1c34544555b6620c3b53f825ea86db
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Citations in the corpus
- Consensus meeting for the management of gastrointestinal stromal tumors. Report of the GIST Consensus Conference of 20-21 March 2004, under the auspices of ESMO. (2005, neutral): “val, under imatinib treatment [40–42].”
- The crucial role of protein phosphorylation in cell signaling and its use as targeted therapy (Review) (2017, neutral): “Alteration of the phosphoproteome also affects gastrointestinal stromal tumors (GISTs) (98,99), lung cancer (73,100), hematologic malignancies (101,102), breast cancer (103,104), pancreatic cancer (105,106) and prostate cancer (103,107).”
- Molecular correlates of imatinib resistance in gastrointestinal stromal tumors. (2006, background): “Conclusion Different molecular mechanisms are responsible for primary and secondary imatinib resistance in GISTs.”
- The structure of Dasatinib (BMS-354825) bound to activated ABL kinase domain elucidates its inhibitory activity against imatinib-resistant ABL mutants. (2006, background): “Interestingly, imatinib, a potent KIT inhibitor and now the standard frontline therapy for advanced gastrointestinal stromal tumors (39, 40) binds to an inactive conformation of KIT (38).”