miR-221 overexpression contributes to liver tumorigenesis

By P. Pineau, Stefano Volinia, K. McJunkin, A. Marchio, C. Battiston, B. Terris, V. Mazzaferro, Scott W. Lowe, Carlo Maria Croce, A. Dejean. Proceedings of the National Academy of Sciences of the United States of America, 2009

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Myc and cell cycle control. (2015, background): “miR-221 is able to induce proliferation targeting p27, both in vitro [165] and in vivo [183].”
Micro‐RNA profiling reveals a role for miR‐29 in human and murine liver fibrosis (2011, background): “Recently, an involvement of miRNAs was demonstrated in highly regulated processes such as hepatocyte apoptosis and hepatocarcinogenesis.(5,6) Furthermore, expression of miR122 correlates with response to interferon treatment of patients infected with hepatitis C virus.”
microRNA-29 can regulate expression of the long non-coding RNA gene MEG3 in hepatocellular cancer (2011, background): “A large and growing body of literature shows the involvement of miRNAs in hepatocarcinogenesis (Meng et al., 2007; Coulouarn et al., 2009; Gramantieri et al., 2009; Ji et al., 2009; Su et al., 2009; Wang et al., 2009; Braconi et al., 2010; Pineau et al., 2010).”
Reprogramming of miRNA networks in cancer and leukemia. (2010, neutral): “, E-TABM-664 (Bloomston et al. 2007), E-TABM-762 and E-TABM-763 (Visone et al. 2009), E-TABM-508 (Pichiorri et al. 2008) , E-TABM-429 (Garzon et al. 2008a), E-TABM-434 (Petrocca et al. 2008), E-TABM-405 (Garzon et al. 2008b), E-TABM-343 (Iorio et al. 2007), E-TABM-41 and E-TABM-42 (Calin et al. 2005”