Allelic replacement in Staphylococcus aureus with inducible counter-selection.
By T. Bae, O. Schneewind. Plasmid, 2006
644 citations recorded by the source corpus.
Subfields: Genetics, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/bd9534fa236fabf52b08a09d05578ca7ea21c41d
How researchers see this paper
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In Genetics
- M. Otto
- A. Peschel
- Victor J. Torres
- S. Kolokotronis
- Susan E Dickey
- Francis Alonzo
- Eric P. Skaar
- Gleb Pishchany
In Molecular Biology
- M. Otto
- Victor J. Torres
- Francis Alonzo
- A. Peschel
- B. Howden
- S. Kolokotronis
- Susan E Dickey
- Ashley L. DuMont
Citations in the corpus
- Staphylococcus aureus Elaborates Leukocidin AB To Mediate Escape from within Human Neutrophils (2013, neutral): “replacement as described previously (33).”
- Reduced Vancomycin Susceptibility in Staphylococcus aureus, Including Vancomycin-Intermediate and Heterogeneous Vancomycin-Intermediate Strains: Resistance Mechanisms, Laboratory Detection, and Clinical Implications (2010, methodology): “(14), and, perhaps most importantly, methods for efficient deliv-”
- Subcellular Localization of the Staphylococcus aureus Heme Iron Transport Components IsdA and IsdB (2009, positive): “The resulting construct was recombined into pKOR1 and used for allelic replacement in Newman (1).”
- Identification of novel cytolytic peptides as key virulence determinants for community-associated MRSA (2007, methodology): “We constructed the hld deletion strains by altering the start codon of the hld gene using the procedure describe”
- Evolution of hypervirulence by a MRSA clone through acquisition of a transposable element (2014, methodology): “Staphylococcus aureus rot::spec mutant strains were generated using the allelic replacement strategy previously described (Bae and Schneewind, 2006).”
- Pathogenicity and virulence of Staphylococcus aureus (2021, background): “lence genes of interest in clinical strains have been developed [282,283] and this should therefore not represent a bottleneck for such research anymore.”
- Cross‐talk between Staphylococcus aureus leukocidins‐intoxicated macrophages and lung epithelial cells triggers chemokine secretion in an inflammasome‐dependent manner (2012, methodology): “Its isogenic pvl mutant (SF8300Dluk-PV), and the complemented strain (SF8300compDluk-PV) in which lukS-PV and lukF-PV were reintroduced into their original chromosome sites in the lukPV mutant by allelic replacement mutagenesis with plasmid pKOR1 (Bae and Schneewind, 2006) have been described previo”
- Characterization of a new cytotoxin that contributes to Staphylococcus aureus pathogenesis (2010, methodology): “Deletion of the lukED and lukAB loci was achieved by allelic replacement as described previously (Bae and Schneewind, 2006).”