What genetics tells us about the causes and mechanisms of Parkinson's disease.

By O. Corti, S. Lesage, A. Brice. Physiological Reviews, 2011

614 citations recorded by the source corpus.

Subfields: Cellular and Molecular Neuroscience, Neurology

View Paper: https://www.semanticscholar.org/paper/bd2b013a8fcafd66b266ca6730cfb73f27f0846f

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1. B. Quintanilla-Vega
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Citations in the corpus

• Molecular mechanisms and physiological functions of mitophagy (2021, neutral): “The relationship between sporadic PD and mitochondrial abnormality has been suggested since 1980s (Corti et al , 2011).”
• Environmental pollutants as risk factors for neurodegenerative disorders: Alzheimer and Parkinson diseases (2015, background): “The identification of genes such as SNCA or PARK1 encoding for α-syn (maybe involved in the regulation of dopamine release and transport), LRRK2 or PARK8 encoding for LRRK2 (or Dardarin), PARK7 encoding for DJ1, PARK6 or PINK1 encoding for PTEN-induced putative kinase 1 (PINK1, a mitochondrial kinas”
• Mitochondrial dysfunction in Parkinson's disease: molecular mechanisms and pathophysiological consequences (2012, neutral): “About 30 pathogenic PINK1 mutations have been identified, among them missense, non-sense, or frameshift mutations, deletions or genomic rearrangements (reviewed in Deas et al , 2009; Nuytemans et al , 2010; and Corti et al , 2011).”