Mechanisms of brain injury after intracerebral haemorrhage.

By G. Xi, R. Keep, J. Hoff. Lancet Neurology, 2006

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Inflammation after Intracerebral Hemorrhage (2007, background): “Increasing evidence suggests that inflammatory mechanisms are involved in ICH-induced brain injury (Aronowski and Hall, 2005; Wang and Tsirka, 2005a; Xi et al, 2006).”
Inhibition of neuronal ferroptosis protects hemorrhagic brain. (2017, neutral): “2003;52(5):1041–1047; discussion 1047–1048.”
Clinical Relevance of Cortical Spreading Depression in Neurological Disorders: Migraine, Malignant Stroke, Subarachnoid and Intracranial Hemorrhage, and Traumatic Brain Injury (2011, background): “Aggravation of brain injury after ICH has been associated with rebleeding, mass effect (Xi et al, 2006), and with brain edema, which develops in a stepwise manner: a transient initial peri-hematoma Journal of Cerebral Blood Flow & Metabolism (2011) 31, 17–35 edema is associated with transient…”
Molecular pathophysiology of cerebral hemorrhage: Secondary brain injury (2011, background): “1 It may occur through many parallel pathological pathways, including: (1) cytotoxicity of blood;2,3 (2) hypermetabolism;4 (3) excitotoxicity;5 (4) spreading depression;6 and (5) oxidative stress and inflammation.”