Phenotypic and functional plasticity of cells of innate immunity: macrophages, mast cells and neutrophils

By S. Galli, N. Borregaard, TA Wynn. Nature Immunology, 2011

1,054 citations recorded by the source corpus.

View Paper: https://www.semanticscholar.org/paper/bad1700275ed9827be3148af853bc0f2da258868

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Citations in the corpus

Satellite Cells and Skeletal Muscle Regeneration (2015, positive): “In the earliest stage after injury the different leukocytes residing in the tissue, for example, mast cells, macrophages and a subset of “patrolling” circulating monocytes, sense the perturbation of tissue homeostasis (14, 107).”
Transition from inflammation to proliferation: a critical step during wound healing (2016, background): “Both infiltrating and skin-resident macrophages are activated by local microenvironmental signals and further develop into various subpopulations, defined by their distinct functional phenotypes [48].”
Phenotypic diversity and plasticity in circulating neutrophil subpopulations in cancer. (2015, background): “In light of the immature state of a subset of LDNs and the mature state of all HDNs, the low- to high-density transition raises the possibility of neutrophil maturation in the circulation, challenging the current paradigm (Galli et al., 2011) and requiring further exploration.”
Wound Healing: A Cellular Perspective. (2019, background): “With the resolution of inflammation, the inflammatory macrophage phenotype transitions into an anti-inflammatory cell type called the alternatively activated macrophage or the M2 macrophage (133).”
Neutrophil cell surface receptors and their intracellular signal transduction pathways (2013, background): “hematopoietic system and share a number of characteristic features with other myeloid cells such as monocytes/macrophages and mast cells [1,2].”