The origins of the developmental origins theory
By D. J. Barker. Journal of Internal Medicine, 2007
2,760 citations recorded by the source corpus.
Subfields: Pediatrics, Perinatology and Child Health
View Paper: https://www.semanticscholar.org/paper/ba54d81c4b39de971c06ca1b5b774677bacb83e2
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In Pediatrics, Perinatology and Child Health
Citations in the corpus
- Pregnancy-specific stress, prenatal health behaviors, and birth outcomes. (2008, background): “Preterm delivery ( 37 weeks) and low birth weight ( 2,500g) are leading causes of infant mortality, infant morbidity, and health problems that may persist into childhood, adolescence and adulthood (Barker, 2007; Goldenberg et al., 1996; Hack & Merkatz, 1995; Mathews & MacDorman, 2006; Newnham, 1998;”
- Instrumental Variable Estimation of the Causal Effect of Hunger Early in Life on Health Later in Life (2016, neutral): “(2005) and Barker (2007) give overviews of the underlying”
- Probiotics and immunity (2009, background): “In addition, diet and exposure to microbes during pregnancy may affect the metabolic and immunological profiles of the pregnant uterus and the risk of developing the disease in the offspring [41].”
- DNA methylation differences after exposure to prenatal famine are common and timing- and sex-specific. (2009, background): “An extensive epidemiological literature has reported associations between characteristics of early development and health outcomes later in life (2,3).”
- Effects of heat stress on mammalian reproduction (2009, background): “physiology during adult life (Barker 2007) is now well accepted, although there is little information about the specific case of maternal heat stress.”
- Developmental Origins of Health and Disease: Brief History of the Approach and Current Focus on Epigenetic Mechanisms (2009, background): “Defects Res A Clin Mol Teratol 2007;79(4):281–288 48.”
- Cell Biology of Ischemia/Reperfusion Injury (2012, background): “Barker and his group (Barker, 1995; 2007) demonstrated that decreasing birth weights over the range from 9 to 5 pounds was associated with increased mortality from ischemic disease in adults.”
- Epigenetics and gene expression (2010, background): “Such long-term changes in gene expression patterns represent an attractive molecular basis for the hypothesis that the origin of adult disease lies in environmental exposure events during an individual’s pre- or post-natal development (Barker, 2007; Heijmans et al., 2008; Szyf, 2009b).”
- National Lipid Association Recommendations for Patient-Centered Management of Dyslipidemia: Part 2. (2015, background): “Much evidence exists to suggest that developmental programming by pathologies during pregnancy influences both fetal development and future cardiometabolic risk of the fetus and the mother.(425,426)”