TRPC6 is a glomerular slit diaphragm-associated channel required for normal renal function

By J. Reiser, K. Polu, Clemens C. Möller, Peter H. Kenlan, M. Altintas, Changli Wei, C. Faul, S. Herbert, I. Villegas, C. Avila-Casado and 8 more. Nature Genetics, 2005

854 citations recorded by the source corpus.

Subfields: Molecular Biology, Nephrology

View Paper: https://www.semanticscholar.org/paper/b9cf42b32fe5500f4a4f53047ebba18d0a24e5c1

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

1. C. Faul
2. P. Mundel
3. M. Donnelly
4. Sandra Merscher-Gomez
5. Y. Chang
6. Stefan E Franz
7. J. Delfgaauw
8. J. Reiser

Citations in the corpus

• The actin cytoskeleton of kidney podocytes is a direct target of the antiproteinuric effect of cyclosporine A (2008, background): “, increased TRPC6-mediated calcium influx may cause FSG”
• TRP Channels (2012, neutral): “from it possible role in heart failure discussed above , there are also indications linking TRPC7 to a mild form autosomal dominant polycystic kidney disease (ADPKD) glutamic acid residue 697.”