Pathogenesis of Helicobacter pylori infection

By M. Gerhard, R. Rad, C. Prinz, M. Naumann. Helicobacter, 2002

2,002 citations recorded by the source corpus.

Subfields: Immunology, Small Animals, Surgery

View Paper: https://www.semanticscholar.org/paper/b786a86eb0b20de38d61de8ebe81b3388d2fa3c9

How researchers see this paper

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In Immunology

1. I. Zilber-Rosenberg
2. J. Oliver
3. Eugene Rosenberg

In Surgery

1. I. Zilber-Rosenberg
2. Eugene Rosenberg

Citations in the corpus

• Liver Fluke Induces Cholangiocarcinoma (2007, background): “Anti-apoptosis has been described in infection-associated cancers such as those caused by Helicobacter pylori [70,71].”
• The Hologenome Concept: Human, Animal and Plant Microbiota (2014, background): “An important property of H. pylori that allows it to be a symbiont in the harsh, acidic environment of the stomach is its ability to secrete urease, which converts urea into ammonia (Kusters et al. 2006).”
• Helicobacter pylori-induced gastric inflammation and gastric cancer. (2014, background): “pylori led to a complete remission in 60–80% of patients with MALT lymphoma [12,13] and a 10-year sustained remission in up to 64% of cases [14].”
• Recent findings on the viable but nonculturable state in pathogenic bacteria. (2010, background): “Another example is H. pylori, which produces gastric and duodenal ulcers, which are among the most widespread and common syndromes in the world (Kusters et al., 2006), and was shown by Adams et al. (2003) to rapidly enter the VBNC FEMS Microbiol Rev 34 (2010) 415–425 c 2009 Federation of European…”
• The return of metabolism: biochemistry and physiology of the pentose phosphate pathway (2014, background): “Moreover, GSH availability, normally high in the stomach, is rapidly depleted after H. pylori infection of the human gastric mucosa, mainly because GSH is used for repairing oxidative DNA lesions in gastric carcinogenesis (Shirin et al., 2001; Basso et al., 2010).”
• Nanomaterials and nanoparticles: Sources and toxicity (2007, background): “Among these diseases are: leukemia (caused by viruses from the Retrovirus and Herpes virus families) [1], cervical cancer (Papilloma virus) [2], liver cancer (Hepatitis virus) [3], gastric ulcer (Helicobacter pylori) [4], nasopharyngeal cancer (Epstein-Barr virus) [5], kidney stones (nanobacteria) [”
• Epidemiology of Gastric Cancer (2015, background): “pylori neutrophil-activating protein (NapA), and proteins encoded by the cag pathogenicity island [34].”
• Lipopolysaccharide modification in Gram-negative bacteria during chronic infection. (2016, background): “…factors of which the vacuolating toxin A (VacA), the cytotoxin-associated gene A (CagA), and LPS play major roles in immunomodulation and contribute to maintain chronic infection (Posselt et al. , 2013, Rubin & Trent, 2013, Chmiela et al. , 2014, de Bernard & Josenhans, 2014, Hatakeyama, 2014).”