Effect of Different Intensities of Exercise on Endothelium-Dependent Vasodilation in Humans: Role of Endothelium-Dependent Nitric Oxide and Oxidative Stress
By Chikara Goto, Y. Higashi, M. Kimura, K. Noma, K. Hara, K. Nakagawa, Mitsutoshi Kawamura, K. Chayama, M. Yoshizumi, I. Nara. Circulation, 2003
656 citations recorded by the source corpus.
Subfields: Cardiology and Cardiovascular Medicine, Complementary and alternative medicine, Physiology
View Paper: https://www.semanticscholar.org/paper/b4c6d306f8cb4d576f02a069b60589e1e6459c50
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In Cardiology and Cardiovascular Medicine
In Complementary and alternative medicine
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Citations in the corpus
- The Impact of High-Intensity Interval Training Versus Moderate-Intensity Continuous Training on Vascular Function: a Systematic Review and Meta-Analysis (2015, result): “Since FMD and CRF (as determined by VO2max or VO2peak) have been shown to be positively associated [45, 46], the result of this meta-analysis is not surprising given the increased ability of HIIT to enhance CRF in patients with cardiometabolic disorders relative to MICT [47].”
- Vascular Adaptation to Exercise in Humans: Role of Hemodynamic Stimuli. (2017, background): “performed an elegant study which randomized subjects to perform 12-week cycle exercise training at mild, moderate or high intensity (90).”
- Exercise, oxidative stress and hormesis. (2008, neutral): “Interestingly, exercise of a very high intensity could also have little effect on vascular function, since the bioavailability of NO can be abolished through scavenging by ROS generated at high intensity exercise (Goto et al., 2003).”
- Effect of exercise training on endothelium‐derived nitric oxide function in humans (2004, background): “Goto et al. (2003) recently studied the effects of 12 weeks of exercise undertaken at low (25% V̇O2max), moderate (50% V̇O2max) and high (75% V̇O2max) intensity in young healthy men.”
- Acute exercise and oxidative stress: a 30 year history (2009, background): “However, because RONS production is known to be a function of both exercise intensity [41] and duration [43], exacerbated prooxidant production that exceeds the currently undefined optimal level, may in turn overwhelm antioxidant defenses in such a way that irreparable oxidative damage may occur, po”