Endoplasmic reticulum stress, obesity and diabetes.

By M. Cnop, F. Foufelle, L. Velloso. Trends in Molecular Medicine, 2012

648 citations recorded by the source corpus.

Subfields: Cell Biology, Physiology, Surgery

View Paper: https://www.semanticscholar.org/paper/b0726f1367264ada889b456291f55cb2ff3aa174

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Citations in the corpus

• A ceramide-centric view of insulin resistance. (2012, background): “Ceramides and ER Stress Endoplasmic reticulum (ER) stress has also been associated with both b cell apoptosis and peripheral and central insulin resistance that contribute to diabetes (Cnop et al., 2012).”
• Palmitic Acid: Physiological Role, Metabolism and Nutritional Implications (2017, background): “Other minor PL are PE (∼5%), phosphatidylinositol (PI) (∼3%), phosphatidylserine (PS), lysoPC, and sphingomyelin (SM); their precise functions are yet to be elucidated (Goerke, 1974; Cockshutt and Possmayer, 1992; Whitsett et al., 2011).”
• Inflammation as a link between obesity, metabolic syndrome and type 2 diabetes. (2014, background): “reticulum stress has been shown in adipose tissue of obese insulin resistant individuals and may be a cause for the development of insulin resistance and inflammation [65].”