Cytotoxic T lymphocytes: all roads lead to death

By M. Barry, R. Bleackley. Nature reviews. Immunology, 2002

978 citations recorded by the source corpus.

Subfields: Immunology

View Paper: https://www.semanticscholar.org/paper/af07c9262c577704aff679fbdf0a1a4c9647bec2

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In Immunology

1. Susan A. Elmore
2. H. Beere

Citations in the corpus

• `The stress of dying': the role of heat shock proteins in the regulation of apoptosis (2004, background): “Once released by cytotoxic T lymphocytes (CTLs) or natural killer (NK) cells into the target cell, granzyme B can directly cleave pro-caspase-3 or Bid to induce apoptosis (Barry and Bleackley, 2002; Pinkoski and Green, 2002; Trapani and Smyth, 2002).”
• Interleukin 12: still a promising candidate for tumor immunotherapy? (2014, neutral): “granule- or FAS-mediated pathways (reviewed in [95]).”
• Apoptosis: A Review of Programmed Cell Death (2007, background): “Reports have also shown that granzyme B can utilize the mitochondrial pathway for amplification of the death signal by specific cleavage of Bid and induction of cytochrome c release (Barry and Bleackley, 2002; Russell and Ley, 2002).”