Methicillin-resistant S. aureus infections among patients in the emergency department.
By G. Moran, A. Krishnadasan, R. Gorwitz, G. Fosheim, L. McDougal, R. Carey, D. Talan. New England Journal of Medicine, 2006
2,321 citations recorded by the source corpus.
Subfields: Epidemiology, Infectious Diseases, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/ae44a36ee880e690dff70006b344feb2e4dd5363
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In Epidemiology
In Infectious Diseases
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In Molecular Biology
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Citations in the corpus
- Hospitalizations and Deaths Caused by Methicillin-Resistant Staphylococcus aureus, United States, 1999–2005 (2007, neutral): “Discussion MRSA, a common cause of nosocomial infections, has emerged as an increasingly common cause of communityassociated infections (20).”
- Clinical practice guidelines by the infectious diseases society of america for the treatment of methicillin-resistant Staphylococcus aureus infections in adults and children: executive summary. (2011, background): “Because of the lack of FDA breakpoints for mupirocin, along with limited availability of commercial tests in the United States, we are unable to provide specific recommendations on mupirocin susceptibility testing for individual patients at this time.”
- A Review of the Research Literature on Evidence-Based Healthcare Design (2008, neutral): “cities (Moran et al., 2006).”
- The epidemic of antibiotic-resistant infections: a call to action for the medical community from the Infectious Diseases Society of America. (2008, neutral): “global pandemic of methicillin-resistant Staphylococcus aureus (MRSA) infection [5-12]; the global spread of”
- Staphylococcus aureus Impairs the Function of and Kills Human Dendritic Cells via the LukAB Toxin (2019, background): “CA-MRSA infections caused by clone USA300, the predominant cause of community-acquired skin infections in the United States (5, 6), frequently recur, indicating that primary infection does not induce protective immunity.”
- Necrotizing soft-tissue infections (2011, background): “Staphylococcus aureus, especially methicillin-resistant species, is increasingly associated with Type II NSTIs, either as a single isolate or in conjunction with streptococci (27, 28).”
- How Staphylococcus aureus biofilms develop their characteristic structure (2012, background): “aureus strains USA300 [Los Angeles County clone (LAC)] and USA400 (MW2) owing to their clinical importance (19, 20), proven involvement in biofilm-associated infections such as osteomyelitis (21) and endocarditis (22), and previously shown dependence of biofilm detachment on the PSM regulator Agr (2”
- Antibiotic resistance and persistence—Implications for human health and treatment perspectives (2020, neutral): “Although most of clinical methicillin-resistant S. aureus (MRSA) isolates are susceptible to rifampicin (Moran et al , 2006), resistance development occurs rapidly if rifampicin is used as monotherapy.”
- Practice guidelines for the diagnosis and management of skin and soft tissue infections: 2014 update by the infectious diseases society of America. (2014, background): “Cutaneous abscesses can be polymicrobial, containing regional skin fl ora or organisms from the adjacent mucous membranes, but S. aureus alone causes a large percentage of skin abscesses, with a substantial number due to MRSA strains [16 – 18].”
- IL-17 is essential for host defense against cutaneous Staphylococcus aureus infection in mice. (2010, background): “Introduction Staphylococcus aureus is a Gram-positive extracellular bacterium that is the most common cause of skin and soft tissue infections, such as cellulitis, impetigo, and folliculitis (1, 2).”