Regulation of Notch signalling by non-visual β-arrestin

By A. Mukherjee, A. Veraksa, A. Bauer, Carine Rossé, J. Camonis, S. Artavanis-Tsakonas. Nature Cell Biology, 2005

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Notch signalling in vertebrate neural development (2006, background): “So, in one tissue, downregulation of Notch could be regulated at a transcriptional level, whereas in another tissue, trafficking of the receptor or the ligand could be the controlling mechanism (for an example, see Ref”
Notch signalling: a simple pathway becomes complex (2006, background): “melanogaster (for example, HRS/VPS27 , Syntaxin I and β-arrestin ) result in elevated Notch protein levels, without affecting the activity of the Notch pathwa”
Notch signaling: simplicity in design, versatility in function (2011, background): “…are not only confined to Wnt, TGF/BMP and hypoxia, but are also elucidated for other pathways, such as the Shh pathway (Driver et al., 2008; Liu et al., 2003; Molnar et al., 2011; Morrow et al., 2009; Mukherjee et al., 2005) and NF-B signaling (for a review, see Poellinger and Lendahl, 2008).”
Notch signaling: the core pathway and its posttranslational regulation. (2009, background): “Deltex physically associates with Notch and the b-arrestin Kurtz, promoting internalization of the tripartite complex and ubiquitination of Notch (Mukherjee et al., 2005).”