Triglyceride-Rich Lipoproteins and Atherosclerotic Cardiovascular Disease: New Insights From Epidemiology, Genetics, and Biology.
By Børge G. Nordestgaard. Circulation Research, 2016
934 citations recorded by the source corpus.
Subfields: Cancer Research, Endocrinology, Diabetes and Metabolism, Surgery
View Paper: https://www.semanticscholar.org/paper/ab99253b687640c654297267290c966256d9dcd0
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Cancer Research
- Børge G. Nordestgaard
- I. Graham
- Klaus Ley
- D. Wolf
- Jan Boren
- Marja-Riitta Taskinen
- Ulrich Laufs
- S. Tsimikas
In Endocrinology, Diabetes and Metabolism
- I. Graham
- Børge G. Nordestgaard
- Jan Boren
- Klaus Ley
- D. Wolf
- Marja-Riitta Taskinen
- M. Chapman
- Alberico L. Catapano
In Surgery
- I. Graham
- Børge G. Nordestgaard
- Jan Boren
- Klaus Ley
- D. Wolf
- M. Chapman
- Marja-Riitta Taskinen
- Alberico L. Catapano
Citations in the corpus
- Triglyceride-rich lipoproteins and their remnants: metabolic insights, role in atherosclerotic cardiovascular disease, and emerging therapeutic strategies—a consensus statement from the European Atherosclerosis Society (2021, methodology): “17,86 These include the remnant-like particle assay 68,158,159 and a direct assay of TRL cholesterol.”
- 2020 International Society of Hypertension global hypertension practice guidelines. (2020, background): “Statins are the lipid-lowering treatment of choice with or without ezetimibe and/or PCSK9 inhibitor (in the optimal setting) [98].”
- Low-density lipoproteins cause atherosclerotic cardiovascular disease: pathophysiological, genetic, and therapeutic insights: a consensus statement from the European Atherosclerosis Society Consensus Panel (2020, background): “and Lp(a) are directly causal in ASCVD, independent of LDL-C levels.(6,7,9,11) Indeed, the hazard ratios for myocardial infarction for a”
- Immunity and Inflammation in atherosclerosis (2019, background): “ApoB-containing triglyceride-rich remnant particles also show a strong association with cardiovascular disease, inflammation, and immune pathways.(7) Indeed, LDL as (auto) antigen was first suggested by Gero et al(38) in 1959: immunization with LDL protected against atherosclerosis in rabbits, sugge”