Reactive species mechanisms of cellular hypoxia-reoxygenation injury.

By Chuanyu Li, R. Jackson. American Journal of Physiology - Cell Physiology, 2002

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Reperfusion injury and reactive oxygen species: The evolution of a concept (2015, background): “Similarities in the in vitro and in vivo responses to I/R (H/R) have been demonstrated using cardiac myocytes [35], intestinal enterocytes [36], alveolar epithelium [34], neurons [37], hepatocytes [38], adipocytes [39], and arterial grafts [40].”
Immune responses of the scallop Chlamys farreri after air exposure to different temperatures (2007, background): “Increased numbers of circulating dead haemocytes following sudden stress is probably due to reduced capacity of surviving haemocytes to remove the debris of dead cells and/or cells swelling and cytolysis induced by oxidative inhibition of membrane Na+–K+ ATPase activity (Li and Jackson, 2002), which”
Free radicals, metals and antioxidants in oxidative stress-induced cancer. (2006, background): “Besides mitochondria, there are other cellular sources of superoxide radical, for example xanthine oxidase (XO), a highly versatile enzyme that is widely distributed among species (from bacteria to man) and within the various tissues of mammals [9].”
The Pharmacological Potential of Rutin (2016, background): “‘Reactive oxygen species’ are identified to participate and play a key task in the pathogenesis of several diseases (Li and Jackson, 2002).”