Bilateral deep brain stimulation in Parkinson's disease: a multicentre study with 4 years follow-up.
By M. Rodríguez-Oroz, Jose A. Obeso, A. Lang, J. Houeto, P. Pollak, S. Rehncrona, J. Kulisevsky, A. Albanese, J. Volkmann, Marwan I. Hariz and 19 more. Brain : a journal of neurology, 2005
1,100 citations recorded by the source corpus.
Subfields: Neurology
View Paper: https://www.semanticscholar.org/paper/a98b2e218640bf556b668cdc2730d6aa7f18416d
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Citations in the corpus
- Functional organization of the basal ganglia: Therapeutic implications for Parkinson's disease (2008, background): “Moreover, it has been convincingly demonstrated in recent years that blockade or lesion of the STN or GPi induced marked clinical improvement and functional restoration of thalamocortical activity in PD patients.(40,41) Chorea-ballism as well as levodopa-induced dyskinesias (LID) in PD are associate”
- Bilateral deep brain stimulation of the pedunculopontine and subthalamic nuclei in severe Parkinson's disease. (2007, background): “In contrast, Rodriguez-Oroz et al. (2005) estimated an additional effect of STN-DBS to the best drug-induced ON-state (about 30% at 1 year observation).”
- Computational analysis of subthalamic nucleus and lenticular fasciculus activation during therapeutic deep brain stimulation. (2006, methodology): “The neuron geometry was defined using Neurolucida (MicroBright- Field, Williston, VT) and converted for display in Rhinoceros and our 3D brain atlas.”
- Deep Brain Stimulation for Parkinson Disease (2010, neutral): “Long-term improvements have been demonstrated for up to 5 years for both STN and GPi DBS, especially on motor fluctuations 45-49 and for tremor with STN, GPi, and ventral intermediate nucleus of the thalamus DBS. 47,50,51 The stimulation parameters in all 3 targets seem to remain relatively stable,…”