Bone resorption by osteoclasts.

By S. Teitelbaum. Science, 2000

4,003 citations recorded by the source corpus.

Subfields: Immunology and Allergy, Molecular Biology, Oncology

View Paper: https://www.semanticscholar.org/paper/a848de03cd8aad01957beca16a3267ac08506cfc

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Immunology and Allergy

1. S. Dorozhkin
2. M. Colonna
3. F. Ross
4. L. Hofbauer
5. Sweta Paliwal
6. S. Ivanovski
7. S. Hienz
8. G. Karsenty

In Molecular Biology

1. S. Teitelbaum
2. F. Ross
3. J. Smolen
4. G. Karsenty
5. S. Dorozhkin
6. M. Colonna
7. L. Hofbauer
8. Sweta Paliwal

In Oncology

1. S. Teitelbaum
2. J. Smolen
3. G. Karsenty
4. S. Dorozhkin
5. M. Colonna
6. F. Ross
7. L. Hofbauer
8. Sweta Paliwal

Citations in the corpus

• Fate decision of mesenchymal stem cells: adipocytes or osteoblasts? (2016, background): “bone formation and resorption results in various diseases, such as osteopetrosis, osteopenia, and osteoporosis.(1) These”
• Mechanisms of Bone Resorption in Periodontitis (2015, background): “It has been shown that multiple mediators, such as IL-1, IL-6, IL-11, IL-17, TNF-α, TNF-beta, TGF-β, kinins, and thrombin, can stimulate bone resorption [4, 7, 13, 23].”
• Osteoimmunology: interplay between the immune system and bone metabolism. (2006, background): “OCs die by apoptotic processes that appear to be regulated by paracrine-acting cytokines and possibly factors in bone matrix (14).”
• Multifunctional Coatings to Simultaneously Promote Osseointegration and Prevent Infection of Orthopaedic Implants (2016, neutral): “Once at the infection site, the macrophages start their own signaling cascade, in combination with bone marrow stromal cells, which eventually leads to differentiation into osteoclasts and hence local bone resorption [144].”
• Role of receptor activator of nuclear factor-κB ligand and osteoprotegerin in bone cell biology (2001, background): “a cell-bound and soluble form, a cell-bound receptor, receptor activator of nuclear factor-κB (RANK), and a secreted decoy receptor, osteoprotegerin (OPG) [4, 5].”
• Glucocorticoid-induced osteoporosis: pathophysiology and therapy (2007, background): “Osteoclasts are members of the monocyte/macrophage family of cells that differentiate under the influence of two requisite cytokines, namely macrophage colony stimulating factor (M-CSF) and receptor activator of NF-κB ligand (RANK-L) [29].”
• FSH directly regulates bone mass. (2006, neutral): “FSH activates three known osteoclastogenic pathways by rapidly enhancing the phosphorylation of Erk1/2, IkBa, and Akt (Teitelbaum, 2000).”
• Osteoclast differentiation and activation (2003, background): “Bone remodelling is the predominant metabolic process regulating bone structure and function during adult life, with the key participant being the osteoclas”
• Making sense of latent TGFβ activation (2003, background): “In vivo, a similar pH is generated by osteoclasts during bone resorption when an integrin-dependent sealing zone is generated between the bone and the cell (Teitelbaum, 2000).”
• Can bioactivity be tested in vitro with SBF solution? (2009, background): “The three main differences between SBF solutions and serum are (i) the absence of proteins, whereas they are known to play an essential role in controlling apatite nucleation (nucleation inhibitors) [10,30]; (ii) the addition of TRIS to buffer SBF solutions, and (iii) the absence of control of the c”