Morphologic and biochemical hallmarks of apoptosis.

By A. Saraste, Kari Pulkki. Cardiovascular Research, 2000

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Apoptosis in cancer: from pathogenesis to treatment (2011, background): “remarkably similar across cell types and species [11,12].”
Chronic palmitate but not oleate exposure induces endoplasmic reticulum stress, which may contribute to INS-1 pancreatic beta-cell apoptosis. (2006, background): “Using a commercial kit that measures cytoplasmic histone-associated DNA fragments, a hallmark of apoptotic cells (29), little if any apoptosis was observed in control cells grown in the presence of serum.”
Apoptosis as anticancer mechanism: function and dysfunction of its modulators and targeted therapeutic strategies (2016, background): “From the morphological point of view apoptotic cells show a characteristic cytoplasmic cell shrinkage, budding of plasma membrane, membrane exposure of phosphatidylserine (PS) on extracellular side, chromatin condensation and DNA fragmentation [8,9].”