Ethanol-induced apoptotic neurodegeneration and fetal alcohol syndrome.

By C. Ikonomidou, P. Bittigau, M. Ishimaru, D. Wozniak, C. Koch, K. Genz, M. Price, V. Stefovska, F. Hörster, T. Tenkova and 2 more. Science, 2000

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Adult neurogenesis: from precursors to network and physiology. (2005, background): “Although these effects of ethanol could be mediated by NMDA or GABAA receptors (224), oxidative stress might also play a role since the antioxidant ebselen, used in the treatment of cognitive disorders of alcoholic patients, prevents ethanol effects on hippocampal neurogenesis (218).”
Early Exposure to Common Anesthetic Agents Causes Widespread Neurodegeneration in the Developing Rat Brain and Persistent Learning Deficits (2003, methodology): “Recent findings indicate that drugs that act by either of these mechanisms induce widespread neuronal apoptosis in immature rat brain when administered during synaptogenesis ( Ikonomidou et al., 1999, 2000; Ishimaru et al., 1999).”
Executive function and the promotion of social–emotional competence (2006, neutral): “…1996), to neuronal migration and differentiation (Crumpton, Atkins, Zawia, & Barone, 2001; Lauder & Schambra, 1999), synapse formation and remodeling (Crnic, 1984; Kleim, Vij, Ballard, & Greenough, 1997; Patrick & Anderson, 2000), and degeneration (Ikonomidou et al., 2000; Ishimaru et al., 1999).”