Human Epicardial Adipose Tissue Is a Source of Inflammatory Mediators

By T. Mazurek, Lifeng Zhang, A. Zalewski, J. Mannion, J. Diehl, H. Arafat, Lea Sarov-Blat, Shawn O’Brien, Elizabeth A Keiper, A. Johnson and 3 more. Circulation, 2003

1,935 citations recorded by the source corpus.

Subfields: Cardiology and Cardiovascular Medicine, Epidemiology, Immunology

View Paper: https://www.semanticscholar.org/paper/a6890170855f3989416cdeda7c85d050df8a85b2

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cardiology and Cardiovascular Medicine

1. H. Willens
2. G. Iacobellis
3. A. Chait
4. Laura J. den Hartigh

In Epidemiology

1. G. Iacobellis
2. A. Chait
3. Laura J. den Hartigh

Citations in the corpus

• Echocardiographic epicardial fat: a review of research and clinical applications. (2009, background): “The presence of inflammatory cells in epicardial adipose tissue could also reflect the response to plaque rupture and lead to the amplification of vascular inflammation and plaque instability.(9) It is also plausible that the paracrine release of cytokines from periadventitial epicardial fat could t”
• Age-related changes in total and regional fat distribution. (2009, neutral): “established antecedents for disease (Mazurek et al., 2003).”
• Adipose Tissue Distribution, Inflammation and Its Metabolic Consequences, Including Diabetes and Cardiovascular Disease (2020, background): “As a metabolically active WAT depot, epiWAT secretes several adipokines and vasoactive substances such as adiponectin, resistin, vascular endothelial growth factor (VEGF), and inflammatory cytokines and chemokines that impact the adjacent myocardium (21).”