Intrauterine growth retardation leads to the development of type 2 diabetes in the rat.

By R. Simmons, Lori Templeton, S. Gertz. Diabetes, 2001

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The Pathophysiology of Gestational Diabetes Mellitus (2018, background): “in utero via bilateral uterine artery ligation [78].”
Metabolic syndrome: definitions and controversies (2011, background): “Evidence from both human [111,112] and animal studies [113,114] suggests that the nutritional, hormonal, and metabolic environment of the mother, as well as the early postnatal environment, may permanently reprogram the structure and physiology of the offspring toward the development of metabolic di”
Severe diabetes, age-dependent loss of adipose tissue, and mild growth deficiency in mice lacking Akt2/PKB beta. (2003, background): “The two groups of mice depicted in Figure 6 may reflect temporal differences in the progression of the phenotype in the population or, conversely, different susceptibilities to development of more extreme diabetes, possibly due to differences in prenatal or perinatal nutrition (37, 38).”