Longitudinal assessment of Pseudomonas aeruginosa in young children with cystic fibrosis.

By J. Burns, R. Gibson, S. Mcnamara, Darlene L. Yim, J. Emerson, M. Rosenfeld, P. Hiatt, K. McCoy, R. Castile, Arnold L. Smith and 1 more. Journal of Infectious Diseases, 2001

624 citations recorded by the source corpus.

Subfields: Molecular Biology, Pulmonary and Respiratory Medicine

View Paper: https://www.semanticscholar.org/paper/a2ea99b0e1ababd7f024f934967a30e3e82ff0b8

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In Molecular Biology

In Pulmonary and Respiratory Medicine

Marin Kollef

Citations in the corpus

Human Toll-like receptor 4 recognizes host-specific LPS modifications (2002, background): “Most children with CF develop PA lung infection by 3 year”
The Pangenome: Diversity, Dynamics and Evolution of Genomes (2020, background): “…aeruginosa, Burkholderia sp., and Haemophilus influenzae have captured microevolution and host adaptation in the human lung (Rau et al. 2012; Lee et al. 2017; Pettigrew et al. 2018; Moleres et al. 2018; Bianconi et al. 2018; Burns et al. 2001; Li et al. 2005; Jorth et al. 2015; Silva et al. 2016).”
Lung Infections Associated with Cystic Fibrosis (2002, background): “(41) suggests that most of this inflammation is a result of prior or concurrent P.”
Pathophysiology and management of pulmonary infections in cystic fibrosis. (2003, background): “aeruginosa infection or lung disease (107, 108) and likely prevent systemic disease.”
Pseudomonas aeruginosa: resistance and therapeutic options at the turn of the new millennium. (2007, background): “Nonmucoid, sensitive strains are often involved initially [135–137] but, without early intervention, irreversible chronic colonisation, most often by mucoid stains, usually occurs within a few months.”
The Epidemiology, Pathogenesis and Treatment of Pseudomonas aeruginosa Infections (2012, background): “aeruginosa by the In one study of patients infected with HIV, the age of 3 years.[38] When chronically infected with P.”