IL-33 amplifies both Th1- and Th2-type responses through its activity on human basophils, allergen-reactive Th2 cells, iNKT and NK cells.

By Molly D. Smithgall, M. Comeau, B. P. Yoon, D. Kaufman, R. Armitage, Dirk E. Smith. International Immunology, 2008

636 citations recorded by the source corpus.

Subfields: Immunology, Physiology, Surgery

View Paper: https://www.semanticscholar.org/paper/9fc1b7003225bbef5ad957c2dd158e4faeb138c4

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In Immunology

1. C. Cayrol
2. J. Girard
3. F. Liew
4. I. McInnes
5. C. A. Dinarello
6. A. Mantovani
7. C. Garlanda

In Surgery

1. C. Cayrol
2. J. Girard
3. F. Liew
4. C. A. Dinarello

Citations in the corpus

• Disease-associated functions of IL-33: the new kid in the IL-1 family (2010, background): “Finally, IL-33 enhances the production of T H 2-type cytokines, including IL-4, and T H 1-type cytokines by natural killer (NK) and NKT cell”
• The IL-1-like cytokine IL-33 is inactivated after maturation by caspase-1 (2009, background): “IL-33 is a potent stimulator of pro-inflammatory cytokine production by mast cells and other cells of the innate immune system (10, 11, 15, 18, 19).”
• The IL-1 family: regulators of immunity (2010, background): “IL-33, despite being associated with T H 2-type responses, induces IFNγ production by NK cells after induction of its receptor by IL-12 (Ref”