Vascular endothelial growth factor ligands and receptors that regulate human cytotrophoblast survival are dysregulated in severe preeclampsia and hemolysis, elevated liver enzymes, and low platelets…
By Yan Zhou, M. Mcmaster, Kirstin Woo, Mary J. Janatpour, J. Perry, Terhi Karpanen, K. Alitalo, C. Damsky, S. Fisher. American Journal of Pathology, 2002
667 citations recorded by the source corpus.
Subfields: Molecular Biology, Obstetrics and Gynecology, Pharmacology
View Paper: https://www.semanticscholar.org/paper/9f088b0e8e92279cc52d14725bef75077a7674a8
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In Obstetrics and Gynecology
Citations in the corpus
- Soluble endoglin contributes to the pathogenesis of preeclampsia (2006, background): “We and others recently showed that high levels of circulating sFlt1 (soluble fms-like tyrosine kinase, also known as soluble VEGF receptor 1) of placental origin may contribute to the pathogenesis of preeclampsi”
- Trophoblast differentiation during embryo implantation and formation of the maternal-fetal interface. (2004, background): “promote cytotrophoblast differentiation and/or invasion and survival (116).”
- Excess placental soluble fms-like tyrosine kinase 1 (sFlt1) may contribute to endothelial dysfunction, hypertension, and proteinuria in preeclampsia. (2003, positive): “In fact, it has recently been shown that exogenous sFlt1 inhibits placental cytotrophoblast invasion in vitro (8).”